Not a class I antiarrhythmic Drug:

Not classified as a Type I antiarrhythmic

Correct match(es) between antiarrhythmic drugs and side effects:

Corneal microdeposits, blurred vision, and photophobia:

Class II anti-arrhythmic drug which also blocks potassium channels:

A patient is diagnosed with atrial fibrillation and exhibits a ventricular following rate of 150 beats / min. Quinidine gluconate (Quinaglute, Quinalan) was administered and although atrial fibrillation was converted to atrial flutter, the ventricular rate increased to 190 beats / min. What is the most likely explanation for the effect on heart rate?

Agent(s) may be effective in terminating paroxysmal supraventricular tachycardia (PSVT)?

Useful in treating third-degree (complete) heart block:

A 70 year old female patient is being treated for ventricular arrhythmias following a myocardial infarction. Runs of ventricular tachyarrythmias, episodes of ventricular fibrillation requiring cardioversion, and pulmonary congestion have been noted. Myocardial ejection fraction is very poor at 25%. Which drugs might be appropriate in this case to suppress these malignant arrhythmias?

Torsades de Pointes, a polymorphic ventricular arrhythmia, is associated with prolongation of Q-T intervals. Which antiarrhythmic agent is most likely to cause this arrhythmia?

Best indicator of digoxin (Lanoxin, Lanoxicaps) efficacy in treating atrial fibrillation:

Cardiac effects resemble those of quinidine gluconate (Quinaglute, Quinalan):

Intravenous lidocaine (Xylocaine):

Most ominous sign of digoxin (Lanoxin, Lanoxicaps) intoxication:

Increases automaticity:

A patient is admitted for treatment of myocardial infarction. During a prolonged recovery period, the patient experienced episodes of vetnricular tachycardia. Following discharge from hospital, the patient was instructed to take a drug to prevent recurrence of ventricular tachycardia. After a few days, the patient complained of dizziness and then lost consciousness. Upon admission to the hospital, the patient was found to be somewhat hypokalemic. A diagnosis of Torsades de Pointes was made. A test to determine the blood level of the antiarrhythmic reveal the drug was present in a slightly subtherapeutic level. What was the drug?

Procainamide (Procan SR, Pronestyl-SR) side effects:

A patient is being treated with digoxin (Lanoxin, Lanoxicaps) for congestive heart failure. To enhance inotropic effects, the dosage was increased. Soon afterwards, the patient complained of dizziness, fatigue, and disturbances in color vision. An ECG was taken. ECG finding(s) consistent with digitalis intoxication include:

A patient is being treated with digoxin for congestive heart failure. To enhance inotropic effects, the dosage was increased. Soon afterwards, the patient complained of dizziness, fatigue, and disturbances in color vision. An ECG was taken. What ECG finding(s) would be consistent with digitalis intoxication?A patient is being treated with digoxin for congestive heart failure. To enhance inotropic effects, the dosage was increased. Soon afterwards, the patient complained of dizziness, fatigue, and disturbances in color vision. An ECG was taken and exhibit arrhythmias consistent with digitialis intoxication.
What antiarrhythmic drugs might be used to treat worsening ventricular arrhythmias with hemodynamic compromise?

Phase of ventricular muscle action potential is associated with an inward,depolarizing Ca2+ current and an outward, repolarizing K+ current.

Automaticity: