Nursing Pharmacology Chapter 26: Renal Pharmacology Practice
Questions
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Acetazolamide (Diamox)
- increases bicarbonate reabsorption in the proximal tubule
- increases secretion of bicarbonate by the ciliary body of the eye into aqueous humor
- may be effective in treatment metabolic alkalosis
- major clincial application is the kidney--to produce a bicarbonate-driven diuresis
- main renal site of action is the thick ascending loop of Henle
Predominate renal location of carbonic acid anhydrase:
- cytoplasm of the epithelial cells
- basolateral membranes
- luminal membrane of the proximal tubule
- A & B
- A, B & C
Acetazolamide (Diamox) secretion:
- not secreted
- excreted by the S2 segment of the proximal tubule
- collecting duct
- distal convoluted tubule
- ascending Loop of Henle
Topically-active carbonic anhydrase inhibitor for ocular use:
- acetazolamide (Diamox)
- furosemide (Lasix)
- chlorothiazide (Diuril)
- dorzolamide (Trusopf)
- bumetanide (Bumex)
Urate stone formation is inhibited at least temporarily by acetazolamide (Diamox) mainly because:
- acetazolamide (Diamox) decreases urine flow
- acetazolamide (Diamox) decreases the amount of urate excreted
- acetazolamide (Diamox) causes an increase in urine pH
- acetazolamide (Diamox) causes a decrease in urine pH
- A & C
Acetazolamide (Diamox) toxicity:
- renal stones
- hyperchloremic metabolic alkalosis
- potassium wasting
- A & C
- A, B & C
Clinical uses of acetazolamide (Diamox):
- urinary alkalinzation
- metabolic alkalosis
- acute mountain sickness
- some forms of epilepsy
- all of the above
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